e2 e2 640 r d systems (R&D Systems)
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R&D Systems
e2 e2 640 r d systems
E2 E2 640 R D Systems, supplied by R&D Systems, used in various techniques. Bioz Stars score: 93/100, based on 18 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/e2+e2+640+r+d+systems/Recombinant+Human%2FMouse%2FRat+UbcH7%2FUBE2L3+Protein%2C+CF/pm41412211-88-27-29
Average 93 stars, based on 18 article reviews
E2 E2 640 R D Systems, supplied by R&D Systems, used in various techniques. Bioz Stars score: 93/100, based on 18 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/e2+e2+640+r+d+systems/Recombinant+Human%2FMouse%2FRat+UbcH7%2FUBE2L3+Protein%2C+CF/pm41412211-88-27-29
Average 93 stars, based on 18 article reviews
e2 e2 640 r d systems - by Bioz Stars,
2026-09
93/100 stars
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In Vitro:Article Title: A TRIM21-UCHL3-ITCH-SIPA1 axis promotes colorectal cancer growth and metastasis. Article Snippet: Dysregulation of protein ubiquitination and deubiquitination is a key mechanism driving tumor progression.. However, the role of upstream regulators and post-translational mechanisms regulating signal-induced proliferation-associated 1 (SIPA1) stability in colorectal cancer (CRC) remains unclear.. This study aimed to investigate novel deubiquitinases that directly bind to and stabilize SIPA1 based on its catalytic activity, along with their underlying action mechanisms. Ubiquitin Proteomics:Article Title: A TRIM21-UCHL3-ITCH-SIPA1 axis promotes colorectal cancer growth and metastasis. Article Snippet: Dysregulation of protein ubiquitination and deubiquitination is a key mechanism driving tumor progression.. However, the role of upstream regulators and post-translational mechanisms regulating signal-induced proliferation-associated 1 (SIPA1) stability in colorectal cancer (CRC) remains unclear.. This study aimed to investigate novel deubiquitinases that directly bind to and stabilize SIPA1 based on its catalytic activity, along with their underlying action mechanisms. Recombinant:Article Title: A TRIM21-UCHL3-ITCH-SIPA1 axis promotes colorectal cancer growth and metastasis. Article Snippet: Dysregulation of protein ubiquitination and deubiquitination is a key mechanism driving tumor progression.. However, the role of upstream regulators and post-translational mechanisms regulating signal-induced proliferation-associated 1 (SIPA1) stability in colorectal cancer (CRC) remains unclear.. This study aimed to investigate novel deubiquitinases that directly bind to and stabilize SIPA1 based on its catalytic activity, along with their underlying action mechanisms. Incubation:Article Title: A TRIM21-UCHL3-ITCH-SIPA1 axis promotes colorectal cancer growth and metastasis. Article Snippet: Dysregulation of protein ubiquitination and deubiquitination is a key mechanism driving tumor progression.. However, the role of upstream regulators and post-translational mechanisms regulating signal-induced proliferation-associated 1 (SIPA1) stability in colorectal cancer (CRC) remains unclear.. This study aimed to investigate novel deubiquitinases that directly bind to and stabilize SIPA1 based on its catalytic activity, along with their underlying action mechanisms. Generated:Article Title: A TRIM21-UCHL3-ITCH-SIPA1 axis promotes colorectal cancer growth and metastasis. Article Snippet: Dysregulation of protein ubiquitination and deubiquitination is a key mechanism driving tumor progression.. However, the role of upstream regulators and post-translational mechanisms regulating signal-induced proliferation-associated 1 (SIPA1) stability in colorectal cancer (CRC) remains unclear.. This study aimed to investigate novel deubiquitinases that directly bind to and stabilize SIPA1 based on its catalytic activity, along with their underlying action mechanisms. |